Search results for "Aortic dissection"

showing 10 items of 109 documents

SGLT-2 (Sodium-Glucose Cotransporter 2) Inhibition Reduces Ang II (Angiotensin II)-Induced Dissecting Abdominal Aortic Aneurysm in ApoE (Apolipoprote…

2019

Objective: Abdominal aortic aneurysm (AAA) is a pathological condition of permanent vessel dilatation that predisposes to the potentially fatal consequence of aortic rupture. SGLT-2 (sodium-glucose cotransporter 2) inhibitors have emerged as powerful pharmacological tools for type 2 diabetes mellitus treatment. Beyond their glucose-lowering effects, recent studies have shown that SGLT-2 inhibitors reduce cardiovascular events and have beneficial effects on several vascular diseases such as atherosclerosis; however, the potential effects of SGLT-2 inhibition on AAA remain unknown. This study evaluates the effect of oral chronic treatment with empagliflozin—an SGLT-2 inhibitor—on dissecting …

0301 basic medicineDissecting Abdominal Aortic AneurysmApolipoprotein EMalemedicine.medical_specialtyInflammation030204 cardiovascular system & hematologyp38 Mitogen-Activated Protein Kinases03 medical and health sciencesMice0302 clinical medicineApolipoproteins EGlucosidesInternal medicinemedicineAnimalsHumansBenzhydryl CompoundsAortic ruptureSodium-Glucose Transporter 2 InhibitorsCells CulturedNeovascularization Pathologicbusiness.industryAngiotensin IINF-kappa Bmedicine.diseaseAngiotensin IIAbdominal aortic aneurysmMatrix MetalloproteinasesMice Inbred C57BLAortic Dissection030104 developmental biologyEndocrinologySodium/Glucose Cotransporter 2Knockout mousemedicine.symptomChemokinesCardiology and Cardiovascular MedicinebusinessAortic Aneurysm AbdominalArteriosclerosis, thrombosis, and vascular biology
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Cardiovascular disease in aging: an overview on thoracic aortic aneurysm as an emerging inflammatory disease

2017

Medial degeneration associated with thoracic aortic aneurysm and acute aortic dissection was originally described by Erdheim as a noninflammatory lesion related to the loss of smooth muscle cells and elastic fibre fragmentation in the media. Recent evidences propose the strong role of a chronic immune/inflammatory process in aneurysm evocation and progression. The coexistence of inflammatory cells with markers of apoptotic vascular cell death in the media of ascending aorta with aneurysms and type A dissections raises the possibility that activated T cells and macrophages may contribute to the elimination of smooth muscle cells and degradation of the matrix. On the other hand, several infla…

0301 basic medicinemedicine.medical_specialtyPathologyAgingImmunologyMyocytes Smooth MuscleThoracic aortic aneurysm; acute aortic dissection; inflammatory pathwayReview Article030204 cardiovascular system & hematologyThoracic aortic aneurysmLesion03 medical and health sciencesAortic aneurysm0302 clinical medicineAneurysmmedicine.arteryInternal medicineThoracic aortic aneurysmAscending aortamedicinelcsh:PathologySettore MED/05 - Patologia ClinicaMyocyteHumansAortic dissectionInflammationAortaacute aortic dissectionAortic Aneurysm Thoracicbusiness.industrySettore MED/23 - Chirurgia CardiacaCell Biologymedicine.diseaseinflammatory pathway030104 developmental biologyCardiovascular DiseasesCardiologycardiovascular systemmedicine.symptombusinesslcsh:RB1-214
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Effect of aneurysm on the mechanical dissection properties of the human ascending thoracic aorta

2012

Objectives: The acute dissection of an ascending thoracic aortic aneurysm (ATAA) represents a devastating separation of elastic layers occurring when the hemodynamic loads on the diseased wall exceed the adhesive strength between layers. At present, the mechanics underlying aortic dissection are largely unclear, and the biomechanical delamination properties of the aneurysmal aorta are not defined. Individuals with bicuspid aortic valve (BAV) are particularly predisposed to ascending aortic aneurysm formation, with a marked risk of aortic dissection. The purpose of this study was to evaluate and compare the dissection properties of nonaneurysmal and aneurysmal human ascending thoracic aorta …

AdultHeart Defects CongenitalMaleAortic valvePulmonary and Respiratory Medicinemedicine.medical_specialtyAorta ThoracicDissection (medical)Risk AssessmentThoracic aortic aneurysmArticleAortic aneurysmBicuspid aortic valveRisk FactorsAneurysm DissectingTensile StrengthInternal medicinemedicine.arterymedicineHumansThoracic aortaAgedAortic dissectionAnalysis of VarianceAortaAortic Aneurysm Thoracicbusiness.industryRisk FactorHemodynamicsSettore ING-IND/34 - Bioingegneria IndustrialeAnatomyMiddle AgedPennsylvaniamedicine.diseaseBiomechanical PhenomenaAortic Dissectionmedicine.anatomical_structureAortic ValveMicroscopy Electron Scanningcardiovascular systemCardiologyFemaleSurgerybusinessCardiology and Cardiovascular MedicineHumanThe Journal of Thoracic and Cardiovascular Surgery
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Incidence of cardiovascular events and risk markers in a prospective study of children diagnosed with Marfan syndrome

2018

Little is known about the incidence of cardiovascular events (CVEs) and their associated risk markers in children with Marfan syndrome (MFS).To assess the incidence of CVEs and determine risk markers in a cohort diagnosed with Marfan syndrome during childhood and followed for several years.From a French multicentre nationwide database, 462 patients with MFS diagnosed during childhood were included prospectively. Patients' files were screened for a period of 20 years (1993-2013). CVEs (e.g. death, aortic dissection, cardiac valve or aortic root surgery) were assessed during the prospective follow-up.Median (interquartile range) age at the end of follow-up was 17.2 (11.1-21.3) years. CVEs wer…

AdultMaleMarfan syndromePediatricsmedicine.medical_specialtyTime FactorsAdolescentDatabases Factual030204 cardiovascular system & hematologyRisk AssessmentMarfan SyndromeYoung Adult03 medical and health sciences0302 clinical medicineRisk FactorsInterquartile rangeCardiac valvemedicineHumansProspective Studies030212 general & internal medicineChildProspective cohort studyAortic dissectionbusiness.industryIncidenceIncidence (epidemiology)Age FactorsInfantGeneral MedicinePrognosismedicine.diseaseConfidence intervalCardiovascular DiseasesChild PreschoolCohortFemaleFranceCardiology and Cardiovascular MedicinebusinessArchives of Cardiovascular Diseases
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Mutations in myosin heavy chain 11 cause a syndrome associating thoracic aortic aneurysm/aortic dissection and patent ductus arteriosus

2006

We have recently described two kindreds presenting thoracic aortic aneurysm and/or aortic dissection ( TAAD) and patent ductus arteriosus (PDA)(1,2) and mapped the disease locus to 16p12.2-p13.13 (ref. 3). We now demonstrate that the disease is caused by mutations in the MYH11 gene affecting the C-terminal coiled-coil region of the smooth muscle myosin heavy chain, a specific contractile protein of smooth muscle cells (SMC). All individuals bearing the heterozygous mutations, even if asymptomatic, showed marked aortic stiffness. Examination of pathological aortas showed large areas of medial degeneration with very low SMC content. Abnormal immunological recognition of SM-MHC and the colocal…

AdultMalePathologymedicine.medical_specialty[SDV]Life Sciences [q-bio]Molecular Sequence DataANEURYSM/DISSECTION030204 cardiovascular system & hematologyBiologyThoracic aortic aneurysmProtein Structure SecondaryDISEASEFamilial thoracic aortic aneurysmCOILED-COILS03 medical and health sciencesAortic aneurysm0302 clinical medicineDuctus arteriosusGeneticsmedicineMYH11LOCUSHumans[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyAmino Acid SequenceDuctus Arteriosus Patent[ SDV.BBM ] Life Sciences [q-bio]/Biochemistry Molecular Biology030304 developmental biologyAortic dissection0303 health sciencesAortic Aneurysm ThoracicBase SequenceMyosin Heavy ChainsSMC proteinHEAVY-CHAIN ISOFORMSAnatomymedicine.diseasePedigreeAortic Dissectionmedicine.anatomical_structureMutationbiology.proteincardiovascular systemFemaleACTA2SMOOTH-MUSCLE MYOSIN
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Analysis of risk factors for neurological dysfunction in patients with acute aortic dissection type A: data from the German Registry for Acute Aortic…

2012

OBJECTIVES: Acute aortic dissection type A (AADA) is associated with major adverse events, such as transient or persistent neurological dysfunction (ND), which may be patient-, disease- or surgery-related. There is a lack of consensus regarding risk factors for ND in AADA patients. The aim of this study was to analyse and identify risk factors for new postoperative ND after aortic repair for AADA. METHODS: Between July 2006 and June 2010, 2137 AADA patients were enrolled in the multi-centre, prospective German Registry of AADA (GERAADA). Perioperative data were prospectively gathered from 50 institutes in Austria, Switzerland and Germany, and multivariate logistic regression analysis was pe…

AdultMalePulmonary and Respiratory Medicinemedicine.medical_specialtyTime FactorsOperative TimeAortic aneurysmPostoperative ComplicationsAneurysmRisk FactorsGermanymedicineHumansRegistriesSurvivorsEmergency TreatmentAgedRetrospective StudiesAortic dissectionAortic Aneurysm Thoracicbusiness.industryIncidencefungiRetrospective cohort studyGeneral MedicineOdds ratioPerioperativeMiddle Agedmedicine.diseaseSurgeryRadiographyAortic DissectionDissectionLogistic ModelsTreatment OutcomeCardiothoracic surgeryAustriaAcute DiseaseMultivariate AnalysisFemaleSurgeryNervous System DiseasesCardiology and Cardiovascular MedicinebusinessVascular Surgical ProceduresSwitzerlandFollow-Up StudiesEuropean Journal of Cardio-Thoracic Surgery
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MFAP5 Loss-of-Function Mutations Underscore the Involvement of Matrix Alteration in the Pathogenesis of Familial Thoracic Aortic Aneurysms and Dissec…

2014

Thoracic aortic aneurysm and dissection (TAAD) is an autosomal-dominant disorder with major life-threatening complications. The disease displays great genetic heterogeneity with some forms allelic to Marfan and Loeys-Dietz syndrome, and an important number of cases still remain unexplained at the molecular level. Through whole-exome sequencing of affected members in a large TAAD-affected family, we identified the c.472CT (p.Arg158(∗)) nonsense mutation in MFAP5 encoding the extracellular matrix component MAGP-2. This protein interacts with elastin fibers and the microfibrillar network. Mutation screening of 403 additional probands identified an additional missense mutation of MFAP5 (c.62GT …

AdultMalecongenital hereditary and neonatal diseases and abnormalitiesAdolescentExtracellular matrix componentNonsense mutationHaploinsufficiencyThoracic aortic aneurysmPathogenesisContractile ProteinsReportGeneticsmedicineHumansMissense mutationGenetics(clinical)ExomeChildGenetics (clinical)AgedGlycoproteinsAged 80 and overGeneticsAortic Aneurysm ThoracicbiologyGenetic heterogeneitySequence Analysis DNAFibroblastsMiddle Agedmedicine.diseasePedigree3. Good healthAortic DissectionAmino Acid SubstitutionCodon Nonsensebiology.proteinIntercellular Signaling Peptides and ProteinsFemaleHaploinsufficiencyElastinThe American Journal of Human Genetics
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Aortic dissection detected by transoesophageal echocardiography

1986

The diagnostic value of transoesophageal echocardiography was evaluated in 24 patients with aortic dissection and compared to transthoracic two-dimensional echocardiography, computer tomography, aortography, surgery and autopsy. Using transoesophageal echocardiography we found in 5 patients a type I dissection, in 5 patients a type II and in 14 patients a type III dissection. Transthoracic two-dimensional echocardiography was positive in 3/5 type I, 2/5 in type II and 2/14 in type III dissections. Computer tomography was unable to demonstrate an intimal flap in 1/2 patients with type I, 2/3 type II and 1/11 type III dissections. Aortography was negative in 1/4 type I, 3/5 type II and 3/12 p…

AdultMalemedicine.medical_specialtyAortographyAutopsyTransoesophageal echocardiographyEsophagusAneurysmmedicine.arterymedicineHumansRadiology Nuclear Medicine and imagingEsophagusCardiac imagingAgedAortic dissectionAortamedicine.diagnostic_testbusiness.industryMiddle Agedmedicine.diseaseAortic AneurysmAortic Dissectionmedicine.anatomical_structureEchocardiographyEvaluation Studies as TopicFemaleRadiologyTomography X-Ray ComputedCardiology and Cardiovascular MedicinebusinessThe International Journal of Cardiac Imaging
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Artery occlusion independently predicts unfavorable outcome in cervical artery dissection

2020

ObjectiveTo assess the impact of dissected artery occlusion (DAO) on functional outcome and complications in patients with cervical artery dissection (CeAD).MethodsWe analyzed combined individual patient data from 3 multicenter cohorts of consecutive patients with CeAD (the Cervical Artery Dissection and Ischemic Stroke Patients [CADISP]–Plus consortium dataset). Patients with data on DAO and functional outcome were included. We compared patients with DAO to those without DAO. Primary outcome was favorable functional outcome (i.e., modified Rankin Scale [mRS] score 0–1) measured 3–6 months from baseline. Secondary outcomes included delayed cerebral ischemia, major hemorrhage, recurrent CeAD…

AdultMalemedicine.medical_specialtyCervical Arterymedicine.medical_treatment030204 cardiovascular system & hematologyRevascularizationArticle03 medical and health sciences0302 clinical medicineInterquartile rangeModified Rankin ScaleAneurysm DissectingInternal medicineHumansMedicineArtery occlusionStrokeAgedbusiness.industryOdds ratioRecovery of FunctionMiddle Agedmedicine.disease3. Good healthAortic DissectionCerebrovascular DisordersDissectionVINTAGECerebrovascular DisorderCardiologyFemale[SDV.SPEE]Life Sciences [q-bio]/Santé publique et épidémiologieCerebral Arterial DiseasesNeurology (clinical)businessCerebral Arterial Disease030217 neurology & neurosurgeryHuman
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Ambulatory follow-up of aortic dissection by transesophageal two-dimensional and color-coded Doppler echocardiography.

1989

Follow-up of 18 patients with aortic dissection (five with type I, one with type II, 11 with type III dissection according to DeBakey) by transesophageal, two-dimensional and color-coded Doppler echocardiography showed a persistence of the false lumen in five of seven patients (71%) after surgery and in nine of 11 patients (82%) after medical therapy. In two patients treated with surgery, the dissected part of the aorta had been resected, whereas in two patients treated medically, a progressive and complete obliteration of the false lumen was observed. In the false lumen, thrombus formation was absent in four, localized in four, and progressive in six patients. Flow within the false lumen c…

AdultMalemedicine.medical_specialtyDissection (medical)Doppler echocardiographyPostoperative ComplicationsAneurysmPhysiology (medical)medicine.arteryAmbulatory CaremedicineHumansThoracic aortaThrombusAortaAgedAortic dissectionAortamedicine.diagnostic_testbusiness.industryMiddle Agedmedicine.diseaseEchocardiography DopplerAortic AneurysmAortic DissectionEchocardiographyRegional Blood FlowDescending aortaFemaleRadiologyCardiology and Cardiovascular MedicinebusinessFollow-Up StudiesCirculation
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